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TWEAK诱导类风湿关节炎成纤维样滑膜细胞合成MMP3的实验研究 |
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| TWEAK诱导类风湿关节炎成纤维样滑膜细胞合成MMP3的实验研究 |
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73(1): 29-36.
［5］ Kamata K, Kamijo S, Nakajima A, et al. Involvement of TNFlike weak inducer of apoptosis in the pathogenesis of collageninduced arthritis［J］. J Immunol, 2006, 177(9): 6433-6439.
［6］ Perper SJ, Browning B, Burkly LC, et al. TWEAK is a novel arthritogenic mediator［J］. J Immunol, 2006, 177(4): 2610-2620.
［7］ Nanki T. Molecular mechanisms of bone destruction in rheumatoid arthritis［J］. Clin Calcium, 2007, 17(4): 510-516.
［8］ Han S, Yoon K, Lee K, et al. TNFrelated weak inducer of apoptosis receptor, a TNF receptor superfamily member, activates NFkappa B through TNF receptorassociated factors［J］. Biochem Biophys Res Commun, 2003, 305(4): 789-796.
［9］ Yepes M, Winkles JA. Inhibition of TWEAK activity as a new treatment for inflammatory and degenerative diseases［J］. Drug News Perspect, 2006 , 19(10): 589-595.上一页 [1] [2] [3] [4] [5] [6] [7] [8] [9] |
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